(Ann Allergy[TA])
5,725 results
  • Objective measurements of compliance in asthma treatment. [Journal Article]
    Ann Allergy. 1994 Dec; 73(6):527-32.Chmelik F, Doughty AAA
  • CONCLUSIONS: Despite an extensive educational program in the self-management of asthma, compliance with recommended treatment was only 40%. Electronic monitoring of peak expiratory flow rate and inhaler usage can provide early identification of patients who do not comply.
  • Immunologic findings in confectionary workers. [Journal Article]
    Ann Allergy. 1994 Dec; 73(6):521-6.Zuskin E, Kanceljak B, … Schachter ENAA
  • CONCLUSIONS: These data suggest that exposure to environmental factors in confectionary plants is associated with frequent respiratory symptoms of an irritative nature. Specific skin testing may be useful in characterizing confectionary workers at risk for the development of occupational asthma.
  • Psyllium hypersensitivity. [Case Reports]
    Ann Allergy. 1994 Dec; 73(6):490-2.Freeman GLAA
  • CONCLUSIONS: The allergens in psyllium appear to be protein in nature and derived from the inner seed endospore and embryo rather than from the husk itself. The patient described herein was sensitized and challenged via ingestion.
  • Cardiac tamponade and recurrent upper respiratory tract infections in a 22-year-old woman. [Clinical Conference]
    Ann Allergy. 1994 Dec; 73(6):473-7.Katial RK, Hatch RM, Baker MRAA
  • The association of IgA deficiency with SLE is clearly established but occurs in only a small percentage of patients. Several hypotheses address the relationship between the two disease processes but the common link remains undetermined. It is important to investigate the diagnosis of IgA deficiency in patients presenting with SLE in order to provide the most appropriate treatment. This patient no…
  • Primary ciliary dyskinesia (the immotile cilia syndrome). [Review]
    Ann Allergy. 1994 Dec; 73(6):457-68; quiz 468-70.Schidlow DVAA
  • CONCLUSIONS: The diagnosis of primary ciliary dyskinesia requires familiarity with the clinical picture and the specific techniques of identification. Although the basic mechanism of disease is known, the molecular genetics of primary ciliary dyskinesia and the causes for the phenotypic variability remain to be explained. Future research should be directed to the identification of the gene(s) responsible for the manifestations of the disease and to effective methods of activation, in vivo, of dysfunctional cilia.