The authors' accepted manuscript, "Mitochondrial Dysfunction promotes Keloid progress through Metabolic Reprogramming: A Narrative Review" [Dermatology. 2026; https://doi.org/10.1159/drm/ackag007] by Lianbo Zhang, Mingxi Li, Guang Zhang, Zichao Wang, and Haiyan Qin has been retracted by the Publisher and the Editor.After peer review, the accepted, unedited manuscript was published online as Early View. Following this online publication, the Journal was notified that the article had previously been published elsewhere [1]. Due to the previous publication and in line with Editorial Policies, the published author's accepted manuscript has been retracted.The authors agree with this retraction.

Objective Maternal diabetes during pregnancy is associated with long-term metabolic and immune-related outcomes in offspring. While psoriasis is strongly linked to metabolic comorbidities later in life, the impact of in utero exposure to maternal diabetes on psoriasis risk in offspring remains unknown. We investigated whether maternal diabetes during pregnancy is associated with the development of childhood psoriasis in offspring. Research Design and Methods We conducted a retrospective population-based cohort study using electronic health record data from Clalit Health Services, Israel's largest health maintenance organization. The cohort included 215,280 singleton live births between 2010 and 2023, with follow-up through 2024. Maternal diabetes during pregnancy was identified using ICD-10 codes and categorized as gestational diabetes, type 1 diabetes, or type 2 diabetes. The primary outcome was childhood psoriasis in offspring. Multivariable Cox proportional hazards analysis adjusted for maternal, pregnancy, and offspring characteristics were used to estimate associations. Results Overall, 5.4% of offspring were born to mothers with diabetes during pregnancy. During follow-up, 1,557 children (0.7%) were diagnosed with psoriasis. Psoriasis prevalence did not differ between offspring exposed and unexposed to maternal diabetes (0.7% vs. 0.7%). No associations were observed by diabetes subtype, treatment modality, or degree of glycemic control. In adjusted analyses, maternal diabetes was not associated with childhood psoriasis in offspring (HR 1.17, 95% CI 0.90-1.54; p = 0.246). Conclusions Maternal diabetes during pregnancy was not associated with increased childhood psoriasis risk in offspring. Our findings do not support a substantial role for prenatal metabolic dysregulation in childhood psoriasis development.

In the article, "Epidemiology of Pruritus: Global Burden of Disease Research from 1990 to 2021 and Future Trend Predictions". [Dermatology. 2025;241(5-6):547-556. https://doi.org/10.1159/000547821] by Wang et al., the authors identified an error in the data used to generate Figure 4. During preparation of the figure, incorrect data from the Global Burden of Disease (GBD) 2021 dataset were used. Specifically, during the generation of the frontier analysis/bibliometric-style figure, the authors inadvertently reused a script that had been configured for an earlier inflammatory bowel disease (IBD) analysis.Fig. 4.Frontier analysis based on the sociodemographic index (SDI) and age-standardized disability-adjusted life years rate (ASDR) due to pruritus. a Frontier analysis based on SDI and pruritus ASDR from 1990 to 2021. b Frontier analysis based on SDI and pruritus ASDR in 2021.The authors have since regenerated Figure 4 and repeated the key analytical steps using datasets from other GBD publications. They reproduced results consistent with those reports (DOIs: 10.1177/20420188241227295 and 10.1097/JS9.0000000000001071), confirming that the data handling procedures, code, and outputs are now functioning as intended.The original sentence read:"The five countries or regions with the largest effective gaps from the frontier included the Netherlands, Germany, Canada, the USA, and Iceland. Conversely, the five countries or regions with the smallest effective gaps included Yemen, Papua New Guinea, Timor-Leste, Lao People's Democratic Republic, and Somalia (Fig. 4)."The corrected sentence should read:"The five countries or regions with the largest effective gaps from the frontier included Taiwan (Province of China), Turkey, Lithuania, Canada, and the United States of America. Conversely, the five countries or regions with the smallest effective gaps included Somalia, Niger, Nepal, Haiti, and Bangladesh (Fig. 4)."The corrected Figure 4 is shown here.

This study conducted a retrospective analysis of 21 patients diagnosed with Collodion Membrane (CM) at a tertiary care center in Riyadh, Saudi Arabia. CM is a rare neonatal condition characterized by a shiny, parchment-like membrane covering the newborn and is associated with genetic disorders related to cornification. Introduction CM is a membrane resembling a parchment that envelops the body of neonates and is frequently associated with genetic epidermal differentiation disorders (EDD). This study aimed to outline the genetic and clinical attributes of CM in Saudi Arabia. Methods This retrospective study was performed at King Abdulaziz Medical City (KAMC) in Riyadh, Saudi Arabia, with approval from the Institutional Review Board of the King Abdullah International Medical Research Center. Informed consent was waived because of the retrospective nature of the study. The participants were patients diagnosed with CM between January 2001 and December 2024. Study data were retrieved from the BestCare® electronic medical record system through chart review. The data collected included demographic information, consanguinity status, duration of NICU stay, clinical features, treatments administered, genetic analyses, and associated conditions. Results 21 patients, of whom 14 were female (66.6%), participated. Consanguinity was documented in all patients (100%). Erythroderma was the most prevalent sign, observed in 12 patients (57.1%). The most frequently identified pathogenic variant was Ceramide Synthase 3 (𝘊𝘌𝘙𝘚3), detected in seven patients (33.3%), while lamellar ichthyosis was the predominant underlying condition, affecting nine patients (42.8%). Conclusion In this cohort of patients with CM, the identified gene mutations were associated with variations in clinical course and outcomes. 𝘊𝘌𝘙𝘚3 mutations were the most frequently detected and were most commonly observed in patients with lamellar ichthyosis, whereas 𝘛𝘎𝘔1 mutations were associated with more severe phenotypes.