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Mitochondrial Proteins journal articles from PubMed MEDLINE database

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Lu Z, Scott I, Webster BR, et al. 
The emerging characterization of lysine residue deacetylation on the modulation of mitochondrial function and cardiovascular biology. [Journal Article, Research Support, N.I.H., Intramural, Review]
Circ Res 2009 Oct 23; 105(9):830-41.
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Vögtle FN, Wortelkamp S, Zahedi RP, et al. 
Global analysis of the mitochondrial N-proteome identifies a processing peptidase critical for protein stability. [Journal Article, Research Support, Non-U.S. Gov't]
Cell 2009 Oct 16; 139(2):428-39.
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Petersen PS, Woldbye DP, Madsen AN, et al. 
In vivo characterization of high Basal signaling from the ghrelin receptor. [Journal Article, Research Support, Non-U.S. Gov't]
Endocrinology 2009 Nov; 150(11):4920-30.
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Jiang D, Zhao L, Clapham DE 
Genome-wide RNAi screen identifies Letm1 as a mitochondrial Ca2+/H+ antiporter. [Journal Article, Research Support, Non-U.S. Gov't]
Science 2009 Oct 2; 326(5949):144-7.
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Demaurex N, Poburko D 
Cell biology. A revolving door for calcium. [Comment, Journal Article, Research Support, Non-U.S. Gov't]
Science 2009 Oct 2; 326(5949):57-8.
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Bak DW, Zuris JA, Paddock ML, et al. 
Redox characterization of the FeS protein mitoNEET and impact of thiazolidinedione drug binding. [JOURNAL ARTICLE]
Biochemistry 2009 Sep 30.
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Chen W, Yang Q, Roeder RG 
Dynamic interactions and cooperative functions of PGC-1alpha and MED1 in TRalpha-mediated activation of the brown-fat-specific UCP-1 gene. [Journal Article, Research Support, N.I.H., Extramural]
Mol Cell 2009 Sep 24; 35(6):755-68.
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Endlicher R, Kriváková P, Lotkova H, et al. 
Tissue specific sensitivity of mitochondrial permeability transition pore to Ca2+ ions. [Journal Article, Research Support, Non-U.S. Gov't]
Acta Medica (Hradec Kralove) 2009; 52(2):69-72.
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Angelini C, Bello L, Spinazzi M, et al. 
Mitochondrial disorders of the nuclear genome. [Journal Article, Research Support, Non-U.S. Gov't]
Acta Myol 2009 Jul; 28(1):16-23.
Abstract | Full Citation
Lamperti C, Zeviani M 
Encephalomyopathies caused by abnormal nuclear-mitochondrial intergenomic cross-talk. [Journal Article]
Acta Myol 2009 Jul; 28(1):2-11.
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