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Potassium Channels, Tandem Pore Domain journal articles from PubMed MEDLINE database

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Barbara G, Alloui A, Nargeot J, et al. 
T-type calcium channel inhibition underlies the analgesic effects of the endogenous lipoamino acids. [Journal Article, Research Support, Non-U.S. Gov't]
J Neurosci 2009 Oct 21; 29(42):13106-14.
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Pang DS, Robledo CJ, Carr DR, et al. 
An unexpected role for TASK-3 potassium channels in network oscillations with implications for sleep mechanisms and anesthetic action. [Journal Article, Research Support, Non-U.S. Gov't]
Proc Natl Acad Sci U S A 2009 Oct 13; 106(41):17546-51.
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Sandoz G, Douguet D, Chatelain F, et al. 
Extracellular acidification exerts opposite actions on TREK1 and TREK2 potassium channels via a single conserved histidine residue. [Journal Article, Research Support, Non-U.S. Gov't]
Proc Natl Acad Sci U S A 2009 Aug 25; 106(34):14628-33.
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Deng PY, Xiao Z, Yang C, et al. 
GABA(B) receptor activation inhibits neuronal excitability and spatial learning in the entorhinal cortex by activating TREK-2 K+ channels. [Journal Article, Research Support, N.I.H., Extramural]
Neuron 2009 Jul 30; 63(2):230-43.
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Nayak TK, Sale H, Nama S, et al. 
Inhibition of human two-pore domain K+ channel TREK1 by local anesthetic lidocaine: negative cooperativity and half-of-the-sites saturation kinetics. [JOURNAL ARTICLE]
Mol Pharmacol 2009 Jul 21.
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Zhou M, Xu G, Xie M, et al. 
TWIK-1 and TREK-1 are potassium channels contributing significantly to astrocyte passive conductance in rat hippocampal slices. [In Vitro, Journal Article, Research Support, Non-U.S. Gov't, Research Support, U.S. Gov't, Non-P.H.S.]
J Neurosci 2009 Jul 1; 29(26):8551-64.
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Dallas ML, Scragg JL, Wyatt CN, et al. 
Modulation of O(2) Sensitive K (+) Channels by AMP-activated Protein Kinase. [Journal Article]
Adv Exp Med Biol 2009.:57-63.
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Wyatt CN, Peers C 
Hetero or homo, hypoxia has them all. [Comment, Journal Article]
J Physiol 2009 Jun 15; 587(Pt 12):2717-8.
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Seifert G, Hüttmann K, Binder DK, et al. 
Analysis of astroglial K+ channel expression in the developing hippocampus reveals a predominant role of the Kir4.1 subunit. [Journal Article, Research Support, Non-U.S. Gov't]
J Neurosci 2009 Jun 10; 29(23):7474-88.
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González JA, Jensen LT, Doyle SE, et al. 
Deletion of TASK1 and TASK3 channels disrupts intrinsic excitability but does not abolish glucose or pH responses of orexin/hypocretin neurons. [Journal Article, Research Support, N.I.H., Extramural, Research Support, Non-U.S. Gov't]
Eur J Neurosci 2009 Jul; 30(1):57-64.
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