| Title | Mechanism of reduction of aortic valvular stenosis by percutaneous transluminal balloon valvuloplasty: report of five cases and review of literature. | | Author(s) | Kennedy KD, Hauck AJ, Edwards WD, Holmes DR, Reeder GS, Nishimura RA | | Institution | Division of Cardiovascular Diseases and Internal Medicine, Mayo Clinic, Rochester, MN 55905. | | Source | Mayo Clin Proc 1988 Aug; 63(8):769-76. | | MeSH | Age Factors
Aged
Aged, 80 and over
Aortic Valve Stenosis
Autopsy
Balloon Dilatation
Calcinosis
Female
Heart Valve Diseases
Humans
Male
| | Abstract | Among five patients (69 to 93 years of age) in whom percutaneous transluminal balloon valvuloplasty was performed for severe aortic stenosis, fractures of cuspid calcium were observed in three, fractures and a cuspid tear in one, and no gross alterations in one. Aortic stenosis resulted from degenerative (senile) calcification of tricuspid aortic valves in two patients, calcification of congenitally bicuspid aortic valves in two, and postinflammatory (presumably rheumatic) fibrocalcific disease in one. Fractures of calcific lesions, by allowing hingelike motion along their sites, seemed to facilitate cuspid mobility and thereby provided the apparent morphologic substrate for reduction in functional stenosis. Among three cases with fused commissures (two bicuspid and one postinflammatory), however, mobility of the conjoined cusps was not achieved after valvuloplasty, despite fracture of calcific nodules in the adjacent valve pocket of one case, because the fused and calcified commissures were not split or fractured and therefore continued to act as rigid struts that impeded cuspid motion. In the current autopsy evaluation of the effects of aortic balloon valvuloplasty, greater cuspid mobility seemed to be achieved in subjects with degenerative calcific stenosis than in those with calcified bicuspid valves or distortion by postinflammatory disease. | | Language | eng | | Pub Type(s) | Case Reports
Journal Article
Review
| | PubMed ID | 3294521 |
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