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Henneke P, Dramsi S, Mancuso G, et al. 
Lipoproteins are critical TLR2 activating toxins in group B streptococcal sepsis. [Journal Article, Research Support, N.I.H., Extramural, Research Support, Non-U.S. Gov't]
J Immunol 2008 May 1; 180(9):6149-58.
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Henneke P, Morath S, Uematsu S, et al. 
Role of lipoteichoic acid in the phagocyte response to group B streptococcus. [Journal Article]
J Immunol 2005 May 15; 174(10):6449-55.
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Machata S, Tchatalbachev S, Mohamed W, et al. 
Lipoproteins of Listeria monocytogenes are critical for virulence and TLR2-mediated immune activation. [Journal Article, Research Support, Non-U.S. Gov't]
J Immunol 2008 Aug 1; 181(3):2028-35.
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Mancuso G, Midiri A, Beninati C, et al. 
Dual role of TLR2 and myeloid differentiation factor 88 in a mouse model of invasive group B streptococcal disease. [Journal Article]
J Immunol 2004 May 15; 172(10):6324-9.
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Henneke P, Takeuchi O, van Strijp JA, et al. 
Novel engagement of CD14 and multiple toll-like receptors by group B streptococci. [Journal Article]
J Immunol 2001 Dec 15; 167(12):7069-76.
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Draper DW, Bethea HN, He YW 
Toll-like receptor 2-dependent and -independent activation of macrophages by group B streptococci. [Journal Article]
Immunol Lett 2006 Feb 15; 102(2):202-14.
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Kenzel S, Mancuso G, Malley R, et al. 
c-Jun kinase is a critical signaling molecule in a neonatal model of group B streptococcal sepsis. [Journal Article]
J Immunol 2006 Mar 1; 176(5):3181-8.
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Baumgärtner M, Kärst U, Gerstel B, et al. 
Inactivation of Lgt allows systematic characterization of lipoproteins from Listeria monocytogenes. [JOURNAL ARTICLE]
J Bacteriol 2006 Oct 13.
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Hashimoto M, Tawaratsumida K, Kariya H, et al. 
Not lipoteichoic acid but lipoproteins appear to be the dominant immunobiologically active compounds in Staphylococcus aureus. [Journal Article]
J Immunol 2006 Sep 1; 177(5):3162-9.
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