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Cen B, Deguchi A, Weinstein IB 
Activation of protein kinase G Increases the expression of p21CIP1, p27KIP1, and histidine triad protein 1 through Sp1. [Journal Article, Research Support, Non-U.S. Gov't]
Cancer Res 2008 Jul 1; 68(13):5355-62.
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Deguchi A, Thompson WJ, Weinstein IB 
Activation of protein kinase G is sufficient to induce apoptosis and inhibit cell migration in colon cancer cells. [Journal Article]
Cancer Res 2004 Jun 1; 64(11):3966-73.
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Deguchi A, Xing SW, Shureiqi I, et al. 
Activation of protein kinase G up-regulates expression of 15-lipoxygenase-1 in human colon cancer cells. [Journal Article]
Cancer Res 2005 Sep 15; 65(18):8442-7.
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Sellak H, Yang X, Cao X, et al. 
Sp1 transcription factor as a molecular target for nitric oxide-- and cyclic nucleotide--mediated suppression of cGMP-dependent protein kinase-Ialpha expression in vascular smooth muscle cells. [Journal Article]
Circ Res 2002 Mar 8; 90(4):405-12.
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Gizard F, Robillard R, Barbier O, et al. 
TReP-132 controls cell proliferation by regulating the expression of the cyclin-dependent kinase inhibitors p21WAF1/Cip1 and p27Kip1. [Journal Article]
Mol Cell Biol 2005 Jun; 25(11):4335-48.
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Hong C, Kim HA, Firestone GL, et al. 
3,3'-Diindolylmethane (DIM) induces a G(1) cell cycle arrest in human breast cancer cells that is accompanied by Sp1-mediated activation of p21(WAF1/CIP1) expression. [Journal Article]
Carcinogenesis 2002 Aug; 23(8):1297-305.
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Adnane J, Bizouarn FA, Qian Y, et al. 
p21(WAF1/CIP1) is upregulated by the geranylgeranyltransferase I inhibitor GGTI-298 through a transforming growth factor beta- and Sp1-responsive element: involvement of the small GTPase rhoA. [Journal Article]
Mol Cell Biol 1998 Dec; 18(12):6962-70.
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Liu L, Li H, Underwood T, et al. 
Cyclic GMP-dependent protein kinase activation and induction by exisulind and CP461 in colon tumor cells. [Journal Article]
J Pharmacol Exp Ther 2001 Nov; 299(2):583-92.
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Huang L, Sowa Y, Sakai T, et al. 
Activation of the p21WAF1/CIP1 promoter independent of p53 by the histone deacetylase inhibitor suberoylanilide hydroxamic acid (SAHA) through the Sp1 sites. [Journal Article]
Oncogene 2000 Nov 23; 19(50):5712-9.
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