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Küchler AM, Pollheimer J, Balogh J, et al. 
Nuclear interleukin-33 is generally expressed in resting endothelium but rapidly lost upon angiogenic or proinflammatory activation. [Journal Article, Research Support, Non-U.S. Gov't]
Am J Pathol 2008 Oct; 173(4):1229-42.
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Zhang H, Issekutz AC 
Down-modulation of monocyte transendothelial migration and endothelial adhesion molecule expression by fibroblast growth factor: reversal by the anti-angiogenic agent SU6668. [Journal Article]
Am J Pathol 2002 Jun; 160(6):2219-30.
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Cattaruzza S, Schiappacassi M, Ljungberg-Rose A, et al. 
Distribution of PG-M/versican variants in human tissues and de novo expression of isoform V3 upon endothelial cell activation, migration, and neoangiogenesis in vitro. [Journal Article]
J Biol Chem 2002 Dec 6; 277(49):47626-35.
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Ristimäki A, Narko K, Enholm B, et al. 
Proinflammatory cytokines regulate expression of the lymphatic endothelial mitogen vascular endothelial growth factor-C. [Journal Article]
J Biol Chem 1998 Apr 3; 273(14):8413-8.
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Thejass P, Kuttan G 
Inhibition of endothelial cell differentiation and proinflammatory cytokine production during angiogenesis by allyl isothiocyanate and phenyl isothiocyanate. [Journal Article]
Integr Cancer Ther 2007 Dec; 6(4):389-99.
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Griffioen AW, Damen CA, Blijham GH, et al. 
Tumor angiogenesis is accompanied by a decreased inflammatory response of tumor-associated endothelium. [Journal Article]
Blood 1996 Jul 15; 88(2):667-73.
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Bussolino F, Ziche M, Wang JM, et al. 
In vitro and in vivo activation of endothelial cells by colony-stimulating factors. [Journal Article]
J Clin Invest 1991 Mar; 87(3):986-95.
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Anderson CR, Hastings NE, Blackman BR, et al. 
Capillary sprout endothelial cells exhibit a CD36 low phenotype: regulation by shear stress and vascular endothelial growth factor-induced mechanism for attenuating anti-proliferative thrombospondin-1 signaling. [Journal Article, Research Support, N.I.H., Extramural]
Am J Pathol 2008 Oct; 173(4):1220-8.
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Aggarwal BB, Totpal K, LaPushin R, et al. 
Diminished responsiveness of senescent normal human fibroblasts to TNF-dependent proliferation and interleukin production is not due to its effect on the receptors or on the activation of a nuclear factor NF-kappa B. [Journal Article]
Exp Cell Res 1995 May; 218(1):381-8.
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