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Poyer F, Coquerel B, Pegahi R, et al. 
Secretion of MMP-2 and MMP-9 induced by VEGF autocrine loop correlates with clinical features in childhood acute lymphoblastic leukemia. [JOURNAL ARTICLE]
Leuk Res 2008 Sep 30.
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Dias S, Hattori K, Zhu Z, et al. 
Autocrine stimulation of VEGFR-2 activates human leukemic cell growth and migration. [Journal Article]
J Clin Invest 2000 Aug; 106(4):511-21.
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Broggini M, Marchini SV, Galliera E, et al. 
Aplidine, a new anticancer agent of marine origin, inhibits vascular endothelial growth factor (VEGF) secretion and blocks VEGF-VEGFR-1 (flt-1) autocrine loop in human leukemia cells MOLT-4. [Journal Article]
Leukemia 2003 Jan; 17(1):52-9.
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Noda K, Ishida S, Shinoda H, et al. 
Hypoxia induces the expression of membrane-type 1 matrix metalloproteinase in retinal glial cells. [Journal Article]
Invest Ophthalmol Vis Sci 2005 Oct; 46(10):3817-24.
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Pegahi R, Poyer F, Legrand E, et al. 
Spontaneous and cytokine-evoked production of matrix metalloproteinases by bone marrow and peripheral blood pre-B cells in childhood acute lymphoblastic leukaemia. [Journal Article]
Eur Cytokine Netw 2005 Sep; 16(3):223-32.
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Dias S, Shmelkov SV, Lam G, et al. 
VEGF(165) promotes survival of leukemic cells by Hsp90-mediated induction of Bcl-2 expression and apoptosis inhibition. [Journal Article]
Blood 2002 Apr 1; 99(7):2532-40.
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Dias S, Hattori K, Heissig B, et al. 
Inhibition of both paracrine and autocrine VEGF/ VEGFR-2 signaling pathways is essential to induce long-term remission of xenotransplanted human leukemias. [Journal Article]
Proc Natl Acad Sci U S A 2001 Sep 11; 98(19):10857-62.
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Ottino P, Finley J, Rojo E, et al. 
Hypoxia activates matrix metalloproteinase expression and the VEGF system in monkey choroid-retinal endothelial cells: Involvement of cytosolic phospholipase A2 activity. [Journal Article]
Mol Vis 2004 May 17.:341-50.
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Dias S, Choy M, Alitalo K, et al. 
Vascular endothelial growth factor (VEGF)-C signaling through FLT-4 (VEGFR-3) mediates leukemic cell proliferation, survival, and resistance to chemotherapy. [Journal Article]
Blood 2002 Mar 15; 99(6):2179-84.
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