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(Papilledema) articles in PubMed
5,746 results
  • Gaze Evoked Deformations of the Peripapillary Retina in Papilledema and Ischemic Optic Neuropathy. [Journal Article]
  • Invest Ophthalmol Vis Sci 2016 Sep 1; 57(11):4979-4987IO
  • Sibony PA
  • CONCLUSIONS: Horizontal eye movements affect the shape of the ppBM layer. The deformation in normals and AION, in adduction, causes posterior displacement temporal to the BMO. In contrast, the deformations in papilledema are larger, involving temporal and nasal sides, presumably because of shifts in cerebrospinal fluid pressure against the scleral flange and hydraulic stiffening of the optic nerve sheath. The clinical importance of gaze-induced deformations is unknown but repetitive motion may be a factor in the genesis or progression of a variety of optic neuropathies.
  • Idiopathic intracranial hypertension following surgical treatment of Cushing disease: case report and review of management strategies. [Journal Article]
  • World Neurosurg 2016 Sep 12WN
  • Wagner J, Fleseriu CM, … Cetas JS
  • CONCLUSIONS: Symptomatic IIH is very rare in adult patients, but can be severe and result in permanent vision loss. A high index of suspicion should be maintained and a fundus exam is necessary to exclude papilledema, whenever there are suggestive symptoms that initially may overlap with AI. It is possible that some cases of mild IIH are misdiagnosed as GC withdrawal and/or AI, however further studies are needed. Treatment consists of re-initiation of higher steroid doses together with acetazolamide with or without cerebrospinal fluid diversion and the priority is to preserve vision and reverse any visual loss.
  • Pseudo-Foster Kennedy Syndrome as a Rare Presentation of Vitamin B12 Deficiency. [Journal Article]
  • Iran Red Crescent Med J 2016; 18(6):e24610IR
  • Petramfar P, Hosseinzadeh F, Mohammadi SS
  • CONCLUSIONS: Demyelinating disease, anterior ischemic optic neuropathy, non-arteritic anterior ischemic optic neuropathy, autoimmune disease, and hereditary optic neuropathy could cause optic neuropathy. Normal CBC parameters and the absence of clinical manifestations of vitamin B12 deficiency could not rule out its diagnosis. Careful physical examinations and history-taking with a classical approach led us to the diagnosis of vitamin B12 deficiency and its treatment.
  • Analysis of Potential Ischemic Effect of Intravitreal Bevacizumab on Unaffected Retina in Treatment-Naïve Macular Edema Due to Branch Retinal Vein Occlusion: A Prospective, Interventional Case-Series. [Journal Article]
  • PLoS One 2016; 11(9):e0162533Plos
  • Rishi P, Raka N, Rishi E
  • CONCLUSIONS: This study analysed the effects with a single injection of bevacizumab. However, whether ischemic adverse effects will emerge with repeated IVB injections as a consequence of cumulative dosing needs further investigation. The setting of our study being a tertiary care centre, the numbers of fresh BRVO cases without prior intervention were limited. Thus, the limitations of our study include a small sample size with a small follow-up period. No major ocular/systemic adverse event was observed in the study period.No evidence of progressive ischaemia attributable to single bevacizumab treatment was observed in this study. However, a larger prospective study involving subjects with cumulative dosing of bevacizumab and a longer follow-up could provide a better understanding of the potential ischaemic effects of bevacizumab or other anti-VEGF agents.
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