Alzheimer Disease
Basics
Description
- Alzheimer disease (AD) is a neurodegenerative condition with insidious inset and progressive impairment of behavioral and cognitive functions.
- AD is the most common type of dementia: 2/3 of cases in >65 years old
- AD is the seventh leading cause of death in the United States.
- People ≥65 years with new AD live 4 to 8 years on average.
- AD is underdiagnosed (~50%), and >50% with AD unaware of diagnosis
- Economic burden in 2023: ~$245 billion, projected $1.1 trillion by 2050 (1)
- Dementia should be distinguished from:
- Age-related cognitive decline: lifelong changes in mental ability and memory; part of normal aging
- Mild cognitive impairment (MCI): decline in cognitive abilities (memory and thinking skills) that is greater than expected for a person’s age but does not interfere significantly with activities of daily living (ADLs).
- MCI: People are generally able to live independently, from a cognitive perspective.
- MCI: affects 17–22% of those ≥65 years, with 32–38% developing dementia ≤5 years (1)
- AD diagnostic classification:
- Preclinical AD: no cognitive symptoms, AD biomarkers present
- MCI due to AD: very mild impairment
- Dementia due to AD:
- Mild: impairment in some activities
- Moderate: impairment in many activities
- Severe: impairment in most activities
- System(s) affected: nervous
- Synonym(s): presenile dementia; senile dementia of the Alzheimer type
Epidemiology
- Predominant age: >65 years
- Incidence: females = males
- Prevalence: females > males
Incidence
New cases of AD in the United States: 484,000/year
- 65 to 75 years: 4 new cases per 1,000 people
- 75 to 84 years: 32 new cases per 1,000 people
- ≥85 years: 76 new cases per 1,000 people
Prevalence
~6.9 million in United States; ~50 million worldwide (1)
- Projected to double in United States by 2050
- 1 in 9 (10.9%) of those aged ≥65 years have AD dementia.
- 33.3% of those aged ≥85 years have AD dementia.
- ~200,000 in United States with early-onset AD (age <65 years)
Etiology and Pathophysiology
- Progressive, irreversible disease, worsening cognitive impairment
- β-Amyloid plaques outside of neurons and τ protein tangles inside of neurons, resulting in loss of connections and neuron death
- Age, genetics, systemic diseases, lifestyle behaviors may influence AD progression.
Genetics
- Autosomal dominant: <5% of AD, usually early onset (age <65 years)
- Familial inheritance AD (non-autosomal dominant): 15–25% of AD
Risk Factors
- Nonmodifiable risk factors (1),(2):
- Age: greatest risk factor, the percentage of people with AD increases dramatically with age
- Family history
- Genetic mutations: in 2022, researchers identified 31 new genes that appear to affect biologic process known to be at play in AD:
- APOE-e4 gene variant: e4 heterozygous 2- to 3-fold risk; e4 homozygous 8-fold risk
- Mutations
- Racial and ethnic differences in AD exist.
- Modifiable risk factors:
- Cardiovascular: hypertension; hyperlipidemia, diabetes, and smoking
- Obesity and diet (Dietary Approaches to Stop Hypertension [DASH], Mediterranean, Mediterranean-DASH Intervention for Neurodegenerative Delay [MIND])
- Lack of physical activity
- Cerebrovascular (stroke) risks and injury
- Other potentially modifiable risk factors:
- Fewer years of formal education (<8th grade)
- Lack of continuous brain activity—learning
- Traumatic brain injuries: repetitive mild and moderate/severe
- Lack of social engagement
- Late-life depression
- Poor quality and inadequate sleep
- Hearing and vision deficits
- High alcohol consumption
- Environmental factors (e.g., air pollution)
General Prevention
- HTN management, increased physical activity, varicella zoster vaccine and cognitive training may delay/prevent cognitive decline, MCI, and AD.
- NSAIDs, estrogen, and vitamin E do not delay AD onset; insufficient evidence for statins and proton pump inhibitors
- Healthy lifestyles may prevent or delay AD.
- Treat psychiatric conditions and avert delirium during hospitalizations.
Commonly Associated Conditions
- Down syndrome
- Depression
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Citation
Domino, Frank J., et al., editors. "Alzheimer Disease." 5-Minute Clinical Consult, 35th ed., Wolters Kluwer, 2027. www.unboundmedicine.com/5minute/view/5-Minute-Clinical-Consult/116014/2.1.0/Alzheimer_Disease.
Alzheimer Disease. In: Domino FJF, Baldor RAR, Golding JJ, et al, eds. 5-Minute Clinical Consult. Wolters Kluwer; 2027. https://www.unboundmedicine.com/5minute/view/5-Minute-Clinical-Consult/116014/2.1.0/Alzheimer_Disease. Accessed July 21, 2026.
Alzheimer Disease. (2027). In Domino, F. J., Baldor, R. A., Golding, J., & Stephens, M. B. (Eds.), 5-Minute Clinical Consult (35th ed.). Wolters Kluwer. https://www.unboundmedicine.com/5minute/view/5-Minute-Clinical-Consult/116014/2.1.0/Alzheimer_Disease
Alzheimer Disease [Internet]. In: Domino FJF, Baldor RAR, Golding JJ, et al, eds. 5-Minute Clinical Consult. Wolters Kluwer; 2027. [cited 2026 July 21]. Available from: https://www.unboundmedicine.com/5minute/view/5-Minute-Clinical-Consult/116014/2.1.0/Alzheimer_Disease.
* Article titles in AMA citation format should be in sentence-case
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5-Minute Clinical Consult

