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[Portal hemodynamics, collateral venous circulation and encephalopathy in patients with esophageal and gastric varices in liver cirrhosis].
Klin Khir. 1998KK

Abstract

Complex hemodynamic investigations were conducted in 166 patients with liver cirrhosis and syndrome of portal hypertension. It was established that gastric varicose veins (VV) in 40% of observations are connected with v. renalis sin. by means of gastrorenal shunts (GRS), esophageal VV in 16.9% of observations--with v. renalis sin. Gastric VV frequently are connected with large GRS. More the GRS diameter, the more pronounced lowering of volemic blood flow in portal vein occurs. While gastric VV presence the volemic blood flow value in portal vein significantly lesser than while isolated esophageal VV. Encephalopathy occurs more frequently in patients with GRS, flowing into v. renalis sin. than in patients while other collateral blood flow ways present. Reverse dependence was revealed between GRS diameter and the degree of portal vein pressure lowering.

Authors

No affiliation info available

Pub Type(s)

Journal Article

Language

rus

PubMed ID

10077911

Citation

Kotenko, O G.. "[Portal Hemodynamics, Collateral Venous Circulation and Encephalopathy in Patients With Esophageal and Gastric Varices in Liver Cirrhosis]." Klinichna Khirurhiia, 1998, pp. 7-9.
Kotenko OG. [Portal hemodynamics, collateral venous circulation and encephalopathy in patients with esophageal and gastric varices in liver cirrhosis]. Klin Khir. 1998.
Kotenko, O. G. (1998). [Portal hemodynamics, collateral venous circulation and encephalopathy in patients with esophageal and gastric varices in liver cirrhosis]. Klinichna Khirurhiia, (11), 7-9.
Kotenko OG. [Portal Hemodynamics, Collateral Venous Circulation and Encephalopathy in Patients With Esophageal and Gastric Varices in Liver Cirrhosis]. Klin Khir. 1998;(11)7-9. PubMed PMID: 10077911.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR T1 - [Portal hemodynamics, collateral venous circulation and encephalopathy in patients with esophageal and gastric varices in liver cirrhosis]. A1 - Kotenko,O G, PY - 1999/3/17/pubmed PY - 1999/3/17/medline PY - 1999/3/17/entrez SP - 7 EP - 9 JF - Klinichna khirurhiia JO - Klin Khir IS - 11 N2 - Complex hemodynamic investigations were conducted in 166 patients with liver cirrhosis and syndrome of portal hypertension. It was established that gastric varicose veins (VV) in 40% of observations are connected with v. renalis sin. by means of gastrorenal shunts (GRS), esophageal VV in 16.9% of observations--with v. renalis sin. Gastric VV frequently are connected with large GRS. More the GRS diameter, the more pronounced lowering of volemic blood flow in portal vein occurs. While gastric VV presence the volemic blood flow value in portal vein significantly lesser than while isolated esophageal VV. Encephalopathy occurs more frequently in patients with GRS, flowing into v. renalis sin. than in patients while other collateral blood flow ways present. Reverse dependence was revealed between GRS diameter and the degree of portal vein pressure lowering. SN - 0023-2130 UR - https://www.unboundmedicine.com/medline/citation/10077911/[Portal_hemodynamics_collateral_venous_circulation_and_encephalopathy_in_patients_with_esophageal_and_gastric_varices_in_liver_cirrhosis]_ L2 - http://www.diseaseinfosearch.org/result/2658 DB - PRIME DP - Unbound Medicine ER -