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Neuroimaging, cognitive, and neurobehavioral outcomes following carbon monoxide poisoning.
Behav Cogn Neurosci Rev 2006; 5(3):141-55BC

Abstract

Carbon monoxide is a colorless, odorless gas produced as a byproduct of combustion. Carbon monoxide is the leading cause of poisoning injury and death worldwide. Morbidity following CO poisoning includes neurologic sequelae, neuropathologic abnormalities on brain imaging, neurobehavioral changes, and cognitive impairments. It is estimated that as high as 50% of individuals with carbon monoxide poisoning will develop neurologic, neurobehavioral, or cognitive sequelae. Carbon monoxide related cognitive impairments included impaired memory, attention, executive function, motor, visual spatial, and slow mental processing speed. Given the high rate of brain related morbidity and the fact that the majority of carbon monoxide is avoidable, awareness and prevention of carbon monoxide poisoning is warranted.

Authors+Show Affiliations

Brigham Young University, USA.No affiliation info available

Pub Type(s)

Journal Article
Review

Language

eng

PubMed ID

16891556

Citation

Hopkins, Ramona O., and Fu Lye M. Woon. "Neuroimaging, Cognitive, and Neurobehavioral Outcomes Following Carbon Monoxide Poisoning." Behavioral and Cognitive Neuroscience Reviews, vol. 5, no. 3, 2006, pp. 141-55.
Hopkins RO, Woon FL. Neuroimaging, cognitive, and neurobehavioral outcomes following carbon monoxide poisoning. Behav Cogn Neurosci Rev. 2006;5(3):141-55.
Hopkins, R. O., & Woon, F. L. (2006). Neuroimaging, cognitive, and neurobehavioral outcomes following carbon monoxide poisoning. Behavioral and Cognitive Neuroscience Reviews, 5(3), pp. 141-55.
Hopkins RO, Woon FL. Neuroimaging, Cognitive, and Neurobehavioral Outcomes Following Carbon Monoxide Poisoning. Behav Cogn Neurosci Rev. 2006;5(3):141-55. PubMed PMID: 16891556.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR T1 - Neuroimaging, cognitive, and neurobehavioral outcomes following carbon monoxide poisoning. AU - Hopkins,Ramona O, AU - Woon,Fu Lye M, PY - 2006/8/8/pubmed PY - 2007/1/16/medline PY - 2006/8/8/entrez SP - 141 EP - 55 JF - Behavioral and cognitive neuroscience reviews JO - Behav Cogn Neurosci Rev VL - 5 IS - 3 N2 - Carbon monoxide is a colorless, odorless gas produced as a byproduct of combustion. Carbon monoxide is the leading cause of poisoning injury and death worldwide. Morbidity following CO poisoning includes neurologic sequelae, neuropathologic abnormalities on brain imaging, neurobehavioral changes, and cognitive impairments. It is estimated that as high as 50% of individuals with carbon monoxide poisoning will develop neurologic, neurobehavioral, or cognitive sequelae. Carbon monoxide related cognitive impairments included impaired memory, attention, executive function, motor, visual spatial, and slow mental processing speed. Given the high rate of brain related morbidity and the fact that the majority of carbon monoxide is avoidable, awareness and prevention of carbon monoxide poisoning is warranted. SN - 1534-5823 UR - https://www.unboundmedicine.com/medline/citation/16891556/Neuroimaging_cognitive_and_neurobehavioral_outcomes_following_carbon_monoxide_poisoning_ L2 - https://medlineplus.gov/carbonmonoxidepoisoning.html DB - PRIME DP - Unbound Medicine ER -