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[Current views on the pathogenesis of migraine aura].
Neurol Neurochir Pol 2007 Jan-Feb; 41(1):70-5NN

Abstract

The pathogenesis of migraine aura, like migraine, remains unclear. The probable cause of migraine aura may be cortical spreading depression (CSD) and cerebral hypoperfusion. Ion changes, activation of the trigeminal nerve and release of neuropeptides seem to be secondary to CSD during an attack of migraine aura. There are many hypotheses of migraine pathogenesis. The focal symptoms during migraine aura may be due to transient constriction of a cerebral artery and headache can result from a sterile inflammatory reaction around the walls of dilated cranial vessels. The development of aura makes a vascular origin a remote possibility, while a primary disturbance of cortical neuron function, probably CSD and activation of the trigeminovascular system, is a more reasonable explanation.

Authors+Show Affiliations

Klinika Neurologii, Akademia Medyczna w Warszawie, Warszawa. domitrz@amwaw.edu.pl

Pub Type(s)

English Abstract
Journal Article
Review

Language

pol

PubMed ID

17330183

Citation

Domitrz, Izabela. "[Current Views On the Pathogenesis of Migraine Aura]." Neurologia I Neurochirurgia Polska, vol. 41, no. 1, 2007, pp. 70-5.
Domitrz I. [Current views on the pathogenesis of migraine aura]. Neurol Neurochir Pol. 2007;41(1):70-5.
Domitrz, I. (2007). [Current views on the pathogenesis of migraine aura]. Neurologia I Neurochirurgia Polska, 41(1), pp. 70-5.
Domitrz I. [Current Views On the Pathogenesis of Migraine Aura]. Neurol Neurochir Pol. 2007;41(1):70-5. PubMed PMID: 17330183.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR T1 - [Current views on the pathogenesis of migraine aura]. A1 - Domitrz,Izabela, PY - 2007/3/3/pubmed PY - 2007/7/18/medline PY - 2007/3/3/entrez SP - 70 EP - 5 JF - Neurologia i neurochirurgia polska JO - Neurol. Neurochir. Pol. VL - 41 IS - 1 N2 - The pathogenesis of migraine aura, like migraine, remains unclear. The probable cause of migraine aura may be cortical spreading depression (CSD) and cerebral hypoperfusion. Ion changes, activation of the trigeminal nerve and release of neuropeptides seem to be secondary to CSD during an attack of migraine aura. There are many hypotheses of migraine pathogenesis. The focal symptoms during migraine aura may be due to transient constriction of a cerebral artery and headache can result from a sterile inflammatory reaction around the walls of dilated cranial vessels. The development of aura makes a vascular origin a remote possibility, while a primary disturbance of cortical neuron function, probably CSD and activation of the trigeminovascular system, is a more reasonable explanation. SN - 0028-3843 UR - https://www.unboundmedicine.com/medline/citation/17330183/[Current_views_on_the_pathogenesis_of_migraine_aura]_ L2 - http://www.diseaseinfosearch.org/result/4811 DB - PRIME DP - Unbound Medicine ER -