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Population prevalence and risk factors for iron deficiency in Auckland, New Zealand.
J Paediatr Child Health. 2007 Jul-Aug; 43(7-8):532-8.JP

Abstract

AIM

Previous prevalence estimates of iron deficiency (ID) in young New Zealand children are inaccurate because of sampling bias and imprecise definition of ID. The aim of this study was to estimate the prevalence of ID in children aged 6-23 months and the factors associated with ID.

METHODS

An ethnically stratified sample identified from random residential address start points. Children resident in Auckland, New Zealand were enrolled from 1999 to 2002. Children with elevated C-reactive protein (>4 mg/L) were excluded. Iron status was determined in 324 (78%) of 416 enrolled children. Analyses adjusted for clustering and weighted for ethnic stratification. ID defined as abnormal values for two or more of serum ferritin (<10 microg/L), iron saturation (<10%) and mean cell volume (<73 fl).

RESULTS

ID was present in 14% (95% confidence interval (CI) 9-17%). ID prevalence varied with ethnicity (Maori 20%, Pacific 17%, other 27%, New Zealand European 7%, P = 0.005), but not with social deprivation. In a multivariate analysis that adjusted for low birthweight, there was an increased risk of ID associated with a body mass index >or= 18.5 kg/m(2) (RR = 4.34, 95% CI 1.08-10.67), and with receiving no infant or follow on formula (RR = 3.60, 95% CI 1.56-6.49).

CONCLUSIONS

ID is prevalent in young New Zealand children. Variance in ID prevalence with ethnicity but not social deprivation implies that cultural practices influence iron status. Young children with more rapid growth are at increased risk of ID as are those receiving milk other than that specifically modified for them.

Authors+Show Affiliations

Department of Paediatrics, University of Auckland, Auckland, New Zealand. cc.grant@auckland.ac.nzNo affiliation info availableNo affiliation info availableNo affiliation info availableNo affiliation info available

Pub Type(s)

Comparative Study
Journal Article
Research Support, Non-U.S. Gov't

Language

eng

PubMed ID

17635681

Citation

Grant, Cameron C., et al. "Population Prevalence and Risk Factors for Iron Deficiency in Auckland, New Zealand." Journal of Paediatrics and Child Health, vol. 43, no. 7-8, 2007, pp. 532-8.
Grant CC, Wall CR, Brunt D, et al. Population prevalence and risk factors for iron deficiency in Auckland, New Zealand. J Paediatr Child Health. 2007;43(7-8):532-8.
Grant, C. C., Wall, C. R., Brunt, D., Crengle, S., & Scragg, R. (2007). Population prevalence and risk factors for iron deficiency in Auckland, New Zealand. Journal of Paediatrics and Child Health, 43(7-8), 532-8.
Grant CC, et al. Population Prevalence and Risk Factors for Iron Deficiency in Auckland, New Zealand. J Paediatr Child Health. 2007 Jul-Aug;43(7-8):532-8. PubMed PMID: 17635681.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR T1 - Population prevalence and risk factors for iron deficiency in Auckland, New Zealand. AU - Grant,Cameron C, AU - Wall,Clare R, AU - Brunt,Deborah, AU - Crengle,Sue, AU - Scragg,Robert, PY - 2007/7/20/pubmed PY - 2008/1/18/medline PY - 2007/7/20/entrez SP - 532 EP - 8 JF - Journal of paediatrics and child health JO - J Paediatr Child Health VL - 43 IS - 7-8 N2 - AIM: Previous prevalence estimates of iron deficiency (ID) in young New Zealand children are inaccurate because of sampling bias and imprecise definition of ID. The aim of this study was to estimate the prevalence of ID in children aged 6-23 months and the factors associated with ID. METHODS: An ethnically stratified sample identified from random residential address start points. Children resident in Auckland, New Zealand were enrolled from 1999 to 2002. Children with elevated C-reactive protein (>4 mg/L) were excluded. Iron status was determined in 324 (78%) of 416 enrolled children. Analyses adjusted for clustering and weighted for ethnic stratification. ID defined as abnormal values for two or more of serum ferritin (<10 microg/L), iron saturation (<10%) and mean cell volume (<73 fl). RESULTS: ID was present in 14% (95% confidence interval (CI) 9-17%). ID prevalence varied with ethnicity (Maori 20%, Pacific 17%, other 27%, New Zealand European 7%, P = 0.005), but not with social deprivation. In a multivariate analysis that adjusted for low birthweight, there was an increased risk of ID associated with a body mass index >or= 18.5 kg/m(2) (RR = 4.34, 95% CI 1.08-10.67), and with receiving no infant or follow on formula (RR = 3.60, 95% CI 1.56-6.49). CONCLUSIONS: ID is prevalent in young New Zealand children. Variance in ID prevalence with ethnicity but not social deprivation implies that cultural practices influence iron status. Young children with more rapid growth are at increased risk of ID as are those receiving milk other than that specifically modified for them. SN - 1034-4810 UR - https://www.unboundmedicine.com/medline/citation/17635681/Population_prevalence_and_risk_factors_for_iron_deficiency_in_Auckland_New_Zealand_ L2 - https://doi.org/10.1111/j.1440-1754.2007.01129.x DB - PRIME DP - Unbound Medicine ER -