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Guillain-Barré syndrome.
Semin Neurol. 2012 Jul; 32(3):179-86.SN

Abstract

Guillain-Barré syndrome (GBS) is an acute inflammatory polyradiculoneuropathy, which has various clinical presentations and both axonal and demyelinating forms. The original description of "ascending paralysis" encompasses the most common varieties: the primary demyelinating form, acute inflammatory demyelinating polyneuropathy (AIDP), and some of the axonal forms, acute motor axonal neuropathy (AMAN) and acute motor and sensory axonal neuropathy (AMSAN). However, there are now well-documented acute "monophasic" polyneuropathies that have a different clinical phenomenology than that described originally by Guillain, Barré, and Strohl: Miller Fisher syndrome, pure sensory neuropathy/neuronopathy, pandysautonomia, and oropharyngeal variant. Here the authors review both typical GBS (AIDP, AMAN, and AMSAN), and variant syndromes with a focus on clinical and diagnostic features, pathologic findings, pathogenesis, and treatment.

Authors+Show Affiliations

Henry Ford Health System Department of Neurology, Detroit, MI, USA. xarcila1@hfhs.orgNo affiliation info available

Pub Type(s)

Journal Article
Review

Language

eng

PubMed ID

23117942

Citation

Arcila-Londono, Ximena, and Richard A. Lewis. "Guillain-Barré Syndrome." Seminars in Neurology, vol. 32, no. 3, 2012, pp. 179-86.
Arcila-Londono X, Lewis RA. Guillain-Barré syndrome. Semin Neurol. 2012;32(3):179-86.
Arcila-Londono, X., & Lewis, R. A. (2012). Guillain-Barré syndrome. Seminars in Neurology, 32(3), 179-86. https://doi.org/10.1055/s-0032-1329196
Arcila-Londono X, Lewis RA. Guillain-Barré Syndrome. Semin Neurol. 2012;32(3):179-86. PubMed PMID: 23117942.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR T1 - Guillain-Barré syndrome. AU - Arcila-Londono,Ximena, AU - Lewis,Richard A, Y1 - 2012/11/01/ PY - 2012/11/3/entrez PY - 2012/11/3/pubmed PY - 2013/4/23/medline SP - 179 EP - 86 JF - Seminars in neurology JO - Semin Neurol VL - 32 IS - 3 N2 - Guillain-Barré syndrome (GBS) is an acute inflammatory polyradiculoneuropathy, which has various clinical presentations and both axonal and demyelinating forms. The original description of "ascending paralysis" encompasses the most common varieties: the primary demyelinating form, acute inflammatory demyelinating polyneuropathy (AIDP), and some of the axonal forms, acute motor axonal neuropathy (AMAN) and acute motor and sensory axonal neuropathy (AMSAN). However, there are now well-documented acute "monophasic" polyneuropathies that have a different clinical phenomenology than that described originally by Guillain, Barré, and Strohl: Miller Fisher syndrome, pure sensory neuropathy/neuronopathy, pandysautonomia, and oropharyngeal variant. Here the authors review both typical GBS (AIDP, AMAN, and AMSAN), and variant syndromes with a focus on clinical and diagnostic features, pathologic findings, pathogenesis, and treatment. SN - 1098-9021 UR - https://www.unboundmedicine.com/medline/citation/23117942/Guillain_Barré_syndrome_ L2 - http://www.thieme-connect.com/DOI/DOI?10.1055/s-0032-1329196 DB - PRIME DP - Unbound Medicine ER -