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[Biochemistry of depression. Literature analysis].
Encephale 1978; 4(3):193-222E

Abstract

The author goes briefly over the metabolism of the main cerebral monoamines, the functioning of synapses, as well as the methods used in studying the biochemistry of depression. Beyond all existing contradictory results, a review of the main works in this field enables us to point out some leading ideas:--Depression would be due to and/or accompanied by a monoaminergic deficiency: some authors emphasize the serotonin one, others the noradrenaline one.--The regulation of mood most probably finds its origin in the monoaminergic balance, rather than in the gross rates of any particular monoamine.--Disturbances are to be found on all metabolic levels: monoaminergic, hydroelectrolytic, hormonal, glucidic, lipidic, lipidic... Close intrication exists between those different metabolisms.--The interaction between the different aminergic systems and the metabolic ways, as well as the dispersion of the acknowledged results, impose more and more the necessity of a biochemical typology of depression, which would lead to a predictive approach to the evolution and treatment of depressive illness.

Authors

No affiliation info available

Pub Type(s)

English Abstract
Journal Article
Review

Language

fre

PubMed ID

30616

Citation

Moussaoui, D. "[Biochemistry of Depression. Literature Analysis]." L'Encephale, vol. 4, no. 3, 1978, pp. 193-222.
Moussaoui D. [Biochemistry of depression. Literature analysis]. Encephale. 1978;4(3):193-222.
Moussaoui, D. (1978). [Biochemistry of depression. Literature analysis]. L'Encephale, 4(3), pp. 193-222.
Moussaoui D. [Biochemistry of Depression. Literature Analysis]. Encephale. 1978;4(3):193-222. PubMed PMID: 30616.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR T1 - [Biochemistry of depression. Literature analysis]. A1 - Moussaoui,D, PY - 1978/1/1/pubmed PY - 1978/1/1/medline PY - 1978/1/1/entrez SP - 193 EP - 222 JF - L'Encephale JO - Encephale VL - 4 IS - 3 N2 - The author goes briefly over the metabolism of the main cerebral monoamines, the functioning of synapses, as well as the methods used in studying the biochemistry of depression. Beyond all existing contradictory results, a review of the main works in this field enables us to point out some leading ideas:--Depression would be due to and/or accompanied by a monoaminergic deficiency: some authors emphasize the serotonin one, others the noradrenaline one.--The regulation of mood most probably finds its origin in the monoaminergic balance, rather than in the gross rates of any particular monoamine.--Disturbances are to be found on all metabolic levels: monoaminergic, hydroelectrolytic, hormonal, glucidic, lipidic, lipidic... Close intrication exists between those different metabolisms.--The interaction between the different aminergic systems and the metabolic ways, as well as the dispersion of the acknowledged results, impose more and more the necessity of a biochemical typology of depression, which would lead to a predictive approach to the evolution and treatment of depressive illness. SN - 0013-7006 UR - https://www.unboundmedicine.com/medline/citation/30616/[Biochemistry_of_depression__Literature_analysis]_ L2 - http://www.diseaseinfosearch.org/result/2199 DB - PRIME DP - Unbound Medicine ER -