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Functional consequences of zinc deficiency.
Prog Food Nutr Sci 1985; 9(1-2):185-226PF

Abstract

Zinc is an essential trace element necessary for over 200 zinc metalloenzymes and required for normal nucleic acid, protein, and membrane metabolism. During the past two decades there has been a rapid expansion of knowledge concerning zinc metabolism in both normal and disease situations, including mechanisms for zinc absorption, excretion and internal redistribution of zinc after stress or trauma. Acrodermatitis enteropathica has been recognized to be a disease of impaired zinc absorption in man. A host of disease processes now are recognized to be complicated by zinc deficiency including alcoholic liver disease, sickle cell anemia, protein calorie malnutrition, and a variety of intestinal diseases including Crohn's disease, sprue, short bowel syndrome and after jejunal ileal bypass. Zinc has proved to be an extremely interesting mineral to nutritionists and physicians because of its importance in normal physiology and biochemistry and because of the diverse presenting features of zinc deficiency. This paper reviews ten functional consequences of zinc deficiency and emphasizes certain consequences in which there have been new discoveries concerning their mechanism (e.g., anorexia) or their clinical importance (e.g., immune dysfunction).

Authors

No affiliation info availableNo affiliation info availableNo affiliation info available

Pub Type(s)

Journal Article
Review

Language

eng

PubMed ID

3911268

Citation

McClain, C J., et al. "Functional Consequences of Zinc Deficiency." Progress in Food & Nutrition Science, vol. 9, no. 1-2, 1985, pp. 185-226.
McClain CJ, Kasarskis EJ, Allen JJ. Functional consequences of zinc deficiency. Prog Food Nutr Sci. 1985;9(1-2):185-226.
McClain, C. J., Kasarskis, E. J., & Allen, J. J. (1985). Functional consequences of zinc deficiency. Progress in Food & Nutrition Science, 9(1-2), pp. 185-226.
McClain CJ, Kasarskis EJ, Allen JJ. Functional Consequences of Zinc Deficiency. Prog Food Nutr Sci. 1985;9(1-2):185-226. PubMed PMID: 3911268.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR T1 - Functional consequences of zinc deficiency. AU - McClain,C J, AU - Kasarskis,E J,Jr AU - Allen,J J, PY - 1985/1/1/pubmed PY - 2001/3/28/medline PY - 1985/1/1/entrez SP - 185 EP - 226 JF - Progress in food & nutrition science JO - Prog Food Nutr Sci VL - 9 IS - 1-2 N2 - Zinc is an essential trace element necessary for over 200 zinc metalloenzymes and required for normal nucleic acid, protein, and membrane metabolism. During the past two decades there has been a rapid expansion of knowledge concerning zinc metabolism in both normal and disease situations, including mechanisms for zinc absorption, excretion and internal redistribution of zinc after stress or trauma. Acrodermatitis enteropathica has been recognized to be a disease of impaired zinc absorption in man. A host of disease processes now are recognized to be complicated by zinc deficiency including alcoholic liver disease, sickle cell anemia, protein calorie malnutrition, and a variety of intestinal diseases including Crohn's disease, sprue, short bowel syndrome and after jejunal ileal bypass. Zinc has proved to be an extremely interesting mineral to nutritionists and physicians because of its importance in normal physiology and biochemistry and because of the diverse presenting features of zinc deficiency. This paper reviews ten functional consequences of zinc deficiency and emphasizes certain consequences in which there have been new discoveries concerning their mechanism (e.g., anorexia) or their clinical importance (e.g., immune dysfunction). SN - 0306-0632 UR - https://www.unboundmedicine.com/medline/citation/3911268/Functional_consequences_of_zinc_deficiency_ DB - PRIME DP - Unbound Medicine ER -