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Ciguatera: current concepts.
J Am Osteopath Assoc. 1995 Mar; 95(3):193-8.JA

Abstract

Ciguatera poisoning develops after ingestion of certain coral reef-associated fish. With travel to and from the tropics and importation of tropical food fish increasing, ciguatera has begun to appear in temperate countries with more frequency. The causative agents are certain varieties of the protozoan dinoflagellate Gambierdiscus toxicus, but bacteria associated with these protozoa may have a role in toxin elaboration. A specific "ciguatoxin" seems to cause the symptoms, but toxicosis may also be a result of a family of toxins. Toxicosis develops from 10 minutes to 30 hours after ingestion of poisoned fish, and the syndrome can include gastrointestinal and neurologic symptoms, as well as chills, sweating, pruritus, bradycardia, tachycardia, and long-lasting weakness and fatigue. More severe features are rare. In this review, the pathophysiologic features and symptoms of ciguatera are reviewed and compared with those of other seafood-related syndromes. Although no definitive therapy is known, the most promising treatment for ciguatera is intravenous administration of mannitol. Physicians should warn patients who are traveling to endemic areas about this toxicosis.

Authors

No affiliation info available

Pub Type(s)

Comparative Study
Journal Article
Review

Language

eng

PubMed ID

7751169

Citation

Levine, D Z.. "Ciguatera: Current Concepts." The Journal of the American Osteopathic Association, vol. 95, no. 3, 1995, pp. 193-8.
Levine DZ. Ciguatera: current concepts. J Am Osteopath Assoc. 1995;95(3):193-8.
Levine, D. Z. (1995). Ciguatera: current concepts. The Journal of the American Osteopathic Association, 95(3), 193-8.
Levine DZ. Ciguatera: Current Concepts. J Am Osteopath Assoc. 1995;95(3):193-8. PubMed PMID: 7751169.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR T1 - Ciguatera: current concepts. A1 - Levine,D Z, PY - 1995/3/1/pubmed PY - 1995/3/1/medline PY - 1995/3/1/entrez SP - 193 EP - 8 JF - The Journal of the American Osteopathic Association JO - J Am Osteopath Assoc VL - 95 IS - 3 N2 - Ciguatera poisoning develops after ingestion of certain coral reef-associated fish. With travel to and from the tropics and importation of tropical food fish increasing, ciguatera has begun to appear in temperate countries with more frequency. The causative agents are certain varieties of the protozoan dinoflagellate Gambierdiscus toxicus, but bacteria associated with these protozoa may have a role in toxin elaboration. A specific "ciguatoxin" seems to cause the symptoms, but toxicosis may also be a result of a family of toxins. Toxicosis develops from 10 minutes to 30 hours after ingestion of poisoned fish, and the syndrome can include gastrointestinal and neurologic symptoms, as well as chills, sweating, pruritus, bradycardia, tachycardia, and long-lasting weakness and fatigue. More severe features are rare. In this review, the pathophysiologic features and symptoms of ciguatera are reviewed and compared with those of other seafood-related syndromes. Although no definitive therapy is known, the most promising treatment for ciguatera is intravenous administration of mannitol. Physicians should warn patients who are traveling to endemic areas about this toxicosis. SN - 0098-6151 UR - https://www.unboundmedicine.com/medline/citation/7751169/Ciguatera:_current_concepts_ L2 - http://jaoa.org/article.aspx?volume=95&page=193 DB - PRIME DP - Unbound Medicine ER -