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[Rheologic changes of blood in portal hypertension].
Khirurgiia (Mosk). 1994 FebK

Abstract

The role of hemorheologic disorders in the pathogenesis of portal hypertension in shown. Rheologic disorders are characteristic of both intrahepatic and extrahepatic forms of portal hypertension. It was found that the rate of hemolysis, disorders of the lipid composition and conformation of red cell proteins is reduced in cirrhosis of the liver complicated by portal hypertension. Changes in the portal system microvessels are attended by microcirculatory disorders which are aggravated by changes in the properties of the cell membrane caused by disorders of the blood physico-chemical properties. Purposeful correction of the hemorheologic disorders has a positive effect on the rheologic parameters only in 3-4 weeks. Postoperative complications occur most frequently on the 10th-14th day. A recurrence of acute hepatic insufficiency is also noted at this time in most patients after drainage of the thoracic duct in aggravation of blood rheology. Direct changes of blood rheology in the portal system were revealed, which is a manifestation of the continuously active mechanism of the syndrome which may cause thromboses and massive hemorrhage.

Authors

Apsatarov EA
No affiliation info available
Andreev GN
No affiliation info available
Liubinskiĭ VL
No affiliation info available
Riudiger PE
No affiliation info available
Mukhamedzhanov GK
No affiliation info available
Ibadildin AS
No affiliation info available

MeSH

AdultErythrocytesFemaleHemodynamicsHemolysisHumansHypertension, PortalLiver CirrhosisPortal SystemPostoperative ComplicationsRecurrenceRheologyTime Factors

Pub Type(s)

Case Reports
English Abstract
Journal Article

Language

rus

PubMed ID

8028244

Citation

Apsatarov, E A., et al. "[Rheologic Changes of Blood in Portal Hypertension]." Khirurgiia, 1994, pp. 33-6.
Apsatarov EA, Andreev GN, Liubinskiĭ VL, et al. [Rheologic changes of blood in portal hypertension]. Khirurgiia (Mosk). 1994.
Apsatarov, E. A., Andreev, G. N., Liubinskiĭ, V. L., Riudiger, P. E., Mukhamedzhanov, G. K., & Ibadildin, A. S. (1994). [Rheologic changes of blood in portal hypertension]. Khirurgiia, (2), 33-6.
Apsatarov EA, et al. [Rheologic Changes of Blood in Portal Hypertension]. Khirurgiia (Mosk). 1994;(2)33-6. PubMed PMID: 8028244.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR T1 - [Rheologic changes of blood in portal hypertension]. AU - Apsatarov,E A, AU - Andreev,G N, AU - Liubinskiĭ,V L, AU - Riudiger,P E, AU - Mukhamedzhanov,G K, AU - Ibadildin,A S, PY - 1994/2/1/pubmed PY - 1994/2/1/medline PY - 1994/2/1/entrez SP - 33 EP - 6 JF - Khirurgiia JO - Khirurgiia (Mosk) IS - 2 N2 - The role of hemorheologic disorders in the pathogenesis of portal hypertension in shown. Rheologic disorders are characteristic of both intrahepatic and extrahepatic forms of portal hypertension. It was found that the rate of hemolysis, disorders of the lipid composition and conformation of red cell proteins is reduced in cirrhosis of the liver complicated by portal hypertension. Changes in the portal system microvessels are attended by microcirculatory disorders which are aggravated by changes in the properties of the cell membrane caused by disorders of the blood physico-chemical properties. Purposeful correction of the hemorheologic disorders has a positive effect on the rheologic parameters only in 3-4 weeks. Postoperative complications occur most frequently on the 10th-14th day. A recurrence of acute hepatic insufficiency is also noted at this time in most patients after drainage of the thoracic duct in aggravation of blood rheology. Direct changes of blood rheology in the portal system were revealed, which is a manifestation of the continuously active mechanism of the syndrome which may cause thromboses and massive hemorrhage. SN - 0023-1207 UR - https://www.unboundmedicine.com/medline/citation/8028244/[Rheologic_changes_of_blood_in_portal_hypertension]_ L2 - https://www.diseaseinfosearch.org/result/5881 DB - PRIME DP - Unbound Medicine ER -