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(Anemia microcytic)
1,776 results
  • Suspecting Hyperferritinemic Sepsis in Iron-Deficient Population: Do We Need a Lower Plasma Ferritin Threshold? [Journal Article]
  • PCPediatr Crit Care Med 2018 May 18
  • Ghosh S, Baranwal AK, … Nallasamy K
  • CONCLUSIONS: Ferritin rises significantly in septic shock patients despite iron deficiency and seems to correlate with the severity of inflammation and organ dysfunction. Even a lower threshold (of 500 or 1,000 ng/mL) could predict higher mortality. It may suggest the need for redefining the plasma ferritin threshold for suspecting hyperferritinemic sepsis and sepsis-induced macrophage activation syndrome in these patients. Larger studies with frequent ferritin measurements are desirable to validate these initial observations.
  • StatPearls [BOOK]
  • BOOKStatPearls Publishing: Treasure Island (FL)
  • Turner Jake J University of Warwick Medical School Bhimji Steve S. SS Un of Toronto
  • Anemia is a condition of low hemoglobin (Hb). It can be subdivided several different ways: symptomatic versus nonsymptomatic, or more frequently, by laboratory findings such as macrocytic versus micr...
  • Clinical Course of Homozygous Hemoglobin Constant Spring in Pediatric Patients. [Journal Article]
  • JPJ Pediatr Hematol Oncol 2018 Apr 17
  • Komvilaisak P, Jetsrisuparb A, … Kiatchoosakun P
  • CONCLUSIONS: Pediatric patients with homozygous Hb Constant Spring developed severe anemia in utero and up to the age of 2 to 3 months postnatal, requiring blood transfusions. Subsequently, their anemia was mild with no evidence of hepatosplenomegaly. Their Hb level was above 9 g/dL with hypochromic microcytic blood pictures as well as wide RDW. Blood transfusions have not been necessary since then.
  • KARS-related diseases: progressive leukoencephalopathy with brainstem and spinal cord calcifications as new phenotype and a review of literature. [Journal Article]
  • OJOrphanet J Rare Dis 2018 Apr 04; 13(1):45
  • Ardissone A, Tonduti D, … Ghezzi D
  • CONCLUSIONS: With our report we define the molecular basis of the previously described Leukoencephalopathy with Brainstem and Spinal cord Calcification widening the spectrum of KARS related disorders, particularly in childhood onset disease suggestive for mitochondrial impairment. The review of previous cases does not suggest a strict and univocal genotype/phenotype correlation for this highly heterogeneous entity. Moreover, our cases confirm the usefulness of search for common brain and spine MR imaging pattern and of broad genetic screening, in syndromes clinically resembling mitochondrial disorders in spite of normal biochemical assay.
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