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(Megaloblastic Anemia)
8,441 results
  • Neurological disorders in vitamin B12 deficiency. [Review]
  • TATer Arkh 2019 May 16; 91(4):122-129
  • Pavlov CS, Damulin IV, … Andreev EA
  • The review discusses thesteps of vitamin B12 metabolism and its role in maintaining of neurological functions. The term "vitamin B12 (cobalamin)" refers to several substances (cobalamins) of a very s…
  • StatPearls: Myelophthisic Anemia [BOOK]
  • BOOKStatPearls Publishing: Treasure Island (FL)
  • Ashorobi Damilola D Nassau University Medical Center Munakomi Sunil S Kathmandu University
  • Anemia is the reduction in the level of hemoglobin, red blood cells (RBCs) or hematocrit below their lower normal range. There are different types and causes of anemia. Anemia subdivides into microcy…
  • StatPearls: Pernicious Anemia [BOOK]
  • BOOKStatPearls Publishing: Treasure Island (FL)
  • Rodriguez Nicole M. NM Aventura Hospital and Medical Center Shackelford Karen S. KS
  • Pernicious anemia (PA) is megaloblastic anemia which results from a deficiency in cobalamin (vitamin B12) due to a deficit of intrinsic factor (IF). Intrinsic factor is a glycoprotein which binds cob…
  • [Hyperpigmentation due to Vitamin B12 Deficiency]. [Journal Article]
  • BNBrain Nerve 2019; 71(4):364-367
  • Araishi K, Nomura Y, Terasaki Y
  • Vitamin B12 deficiency is associated with mucocutaneous symptoms, such as glossitis and hyperpigmentation, as well as megaloblastic anemia and neuropathy. These are reversible conditions, and the sym…
  • Identification of an iron-responsive subtype in two children diagnosed with relapsing-remitting multiple sclerosis using whole exome sequencing. [Journal Article]
  • MGMol Genet Metab Rep 2019; 19:100465
  • van Rensburg SJ, Peeters AV, … Kotze MJ
  • CONCLUSIONS: Our findings highlight the potential value of WES to identify heritable risk factors that could affect the reabsorption of transferrin-bound iron in the kidneys causing sustained iron loss, together with inhibition of vitamin B12 absorption and vitamin D reabsorption (CUBN) and iron transport into mitochondria (SLC25A37) as the sole site of heme synthesis. This supports a model for RRMS in children with an apparent iron-deficient biochemical subtype of MS, with oligodendrocyte cell death and impaired myelination possibly caused by deficits of energy- and antioxidant capacity in mitochondria.
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