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(cochleitis)
10 results
  • Endocochlear inflammation in cochlear implant users: case report and literature review. [Case Reports]
    Int J Pediatr Otorhinolaryngol 2013; 77(6):885-93Benatti A, Castiglione A, … Martini A
  • CONCLUSIONS: Despite efforts by both surgeons and manufacturers, device-related and surgical complications still occur. These and other rare conditions demand specific management, and their frequency may be underestimated. Further studies are needed to assess more realistic rates of complications and devise more efficient strategies for early diagnosis and treatment.
  • Auditory nerve function following cochleitis. [Case Reports]
    Int J Pediatr Otorhinolaryngol 2012; 76(11):1696-701Levi J, Looney L, … Morlet T
  • We present a case of temporary cochlear nerve dysfunction due to endocochlear inflammation with subsequent recovery. Retrospective case review at a pediatric tertiary care hospital. A nine-year-old male presented seven years post-cochlear implantation with an electrode array extruded into the external auditory canal. Upon exploration in the operating room, turbid perilymph from the scala tympani …
  • Pathology and pathophysiology of idiopathic sudden sensorineural hearing loss. [Case Reports]
    Otol Neurotol 2005; 26(2):151-60Merchant SN, Adams JC, Nadol JB
  • CONCLUSIONS: The temporal bone findings do not support the concept of membrane breaks, perilymphatic fistulae, or vascular occlusion as common causes for idiopathic sudden sensorineural hearing loss. The finding in our one case acquired acutely during idiopathic sudden sensorineural hearing loss as well as other clinical and experimental observations do not strongly support the theory of viral cochleitis.We put forth the hypothesis that idiopathic sudden sensorineural hearing loss may be the result of pathologic activation of cellular stress pathways involving nuclear factor-kappaB within the cochlea.
  • [Acute sensorineural hearing loss at the Otorhinolaryngology Department of the General Hospital in Subotica 1991-1996]. [Journal Article]
    Med Pregl 1999 Jan-Feb; 52(1-2):44-52Rakić N
  • CONCLUSIONS: The study included 53 patients, 27 (50.94%) female and 26 (49.06%) males (Graph 1). Their age (Graph 2) ranged between 16-71 years. The biggest group of patients was between 40-49 (39.62%) years of age. Distribution per year (Graph 3) shows that the incidence of the acute sensorineural hearing loss differs from the findings of other authors, especially in the period of 1991-1994, due to very few cases. Connection of this illness with seasons is shown in Graph 4. We had most cases during summer time (August, 16.98%). There is a mild rise in winter (December, 13.21%). The degree of the hearing loss in decibels is shown in the Graph 5. Most of the patients (75.47%) had mild or severely damaged hearing (40-85 dB). Period of the deafness before patients were admitted to the hospital, is shown in Graph 7. Most of them came in the first seven days after they experienced acute deafness (52.83%). Symptomatology of the persons with acute hearing loss is shown in Table 1. All of them had the feeling of deafness, and 50.94% also had tinnitus. Table 2 shows that only two patients had a pathologic finding (hypo function) on the vestibular caloric test. 57.14% of patients tested by this method showed a normal function of the vestibular apparatus. The recovery of he
  • The pathology of idiopathic sudden sensorineural hearing loss. [Case Reports]
    Arch Otorhinolaryngol 1986; 243(1):1-15Schuknecht HF, Donovan ED
  • We examined the temporal bone pathologies in 12 ears with idiopathic sudden sensorineural hearing loss, and found that the lesions present in these specimens and in 10 others reported in the literature are similar to lesions occurring in known cases of viral cochleitis. These lesions are unlike those resulting from known vascular causes.
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