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(hypophosphatemic)
2,003 results
  • Oncogenic osteomalacia caused by a phosphaturic mesenchymal tumor of the femur: A case report. [Case Reports]
    World J Clin Cases 2019; 7(15):2081-2086Tang D, Wang XM, … Mi XX
  • CONCLUSIONS: Diagnosis of oncogenic osteomalacia is difficult due to the varieties and localization of source tumors and absence of pathognomonic biomedical signs. Our case highlights the importance of a combination of medical imaging and biopsy in the diagnosis of oncogenic osteomalacia caused by a phosphaturic mesenchymal tumor.
  • Mineralized tissues in hypophosphatemic rickets. [Journal Article]
    Pediatr Nephrol 2019Robinson ME, AlQuorain H, … Rauch F
  • Hypophosphatemic rickets is caused by renal phosphate wasting that is most commonly due to X-linked dominant mutations in PHEX. PHEX mutations cause hypophosphatemia indirectly, through the increased expression of fibroblast growth factor 23 (FGF23) by osteocytes. FGF23 decreases renal phosphate reabsorption and thereby increases phosphate excretion. The lack of phosphate leads to a mineralizatio…
  • [Tumor-induced Hypophosphatemic Osteomalacia Caused by a Chest Wall Tumor]. [Case Reports]
    Kyobu Geka 2019; 72(8):570-573Kin Y, Ogawa H, … Maniwa Y
  • Tumor-induced osteomalacia (TIO) is a rare paraneoplastic disease characterized by hypophosphatemia and skeletal undermineralization. Overproduction of fibroblast growth factor 23(FGF23) from the responsible tumor is reported to be a causative factor. Removing the tumor is the only effective treatment for TIO, but identifying the tumor is sometimes difficult. A 43-year-old man complained of heel …
  • Phosphate matters when investigating hypercalcemia: a mutation in SLC34A3 causing HHRH [Journal Article]
    Endocrinol Diabetes Metab Case Rep 2019; 2019(1):1-6Tang AR, Hinz LE, … Kline GA
  • Hereditary hypophosphatemic rickets with hypercalciuria (HHRH) is a rare, autosomal recessive disorder caused by mutations in the SLC34A3 gene that encodes the renal sodium-dependent phosphate cotransporter 2c (NaPi-IIc). It may present as intermittent mild hypercalcemia which may attract initial diagnostic attention but appreciation of concomitant hypophosphatemia is critical for consideration o…
  • Fibroblast growth factor 23 and phosphate homeostasis. [Journal Article]
    Curr Opin Nephrol Hypertens 2019; 28(5):465-473Balani S, Perwad F
  • CONCLUSIONS: Significant advances in understanding the cellular and molecular aspects of renal tubular phosphate transport and its regulation by FGF23 has led to the discovery of novel therapeutics to treat human disorders of phosphate homeostasis.
  • Aberrant activation of Wnt signaling pathway altered osteocyte mineralization. [Journal Article]
    Bone 2019; 127:324-333Zhou Y, Lin J, … Xiao Y
  • Mineralization of bone is a dynamic process, involving a complex interplay between cells, secreted macromolecules, signaling pathways, and enzymatic reactions; the dysregulation of bone mineralization may lead to serious skeletal disorders, including hypophosphatemic rickets, osteoporosis, and rheumatoid arthritis. Very few studies have reported the role of osteocytes - the most abundant bone cel…
  • Management of phosphate abnormalities in hemodialysis patients: Findings from Malaysia. [Journal Article]
    Saudi J Kidney Dis Transpl 2019 May-Jun; 30(3):670-677Mohamed Koya SNM
  • Studies have shown that the mean or median phosphate levels were related to certain factors although applying this finding into the clinical setting is challenging. In this study, we attempted to determine treatment characteristics for patients with end-stage renal disease (ESRD) on maintenance hemodialysis (MHD) having hyperphosphatemia or hypophosphatemia in comparison with those with normal ph…
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