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(vasoconstrictive)
3,303 results
  • Novel Medications for the Treatment of Migraine. [Review]
    Headache 2019; 59(9):1597-1608Ceriani CEJ, Wilhour DA, Silberstein SD
  • CONCLUSIONS: The development of new migraine-specific classes of medications provides more treatment options for both acute and preventive treatment of migraine.
  • The role of arginine vasopressin in myocardial infarction and reperfusion. [Journal Article]
    Kardiol Pol 2019Nobian A, Ashfaq M, Spyridopoulos I
  • Little attention is paid to the coronary microvasculature when treating acute myocardial infarction (MI). Microvascular obstruction (MVO) contributes to ischaemia-reperfusion (I-R) injury which hampers distal blood flow to the myocardium despite recanalisation of the culprit epicardial vessel. One of the mechanisms behind reperfusion injury is MVO due to persistent vasoconstrictor tone during rep…
  • [Cognitive impairments and microvascular endothelial dysfunction in unilateral occlusion of the carotid artery]. [Journal Article]
    Angiol Sosud Khir 2019; 25(3):17-22Guliaev SM, Urbanova EZ
  • CONCLUSIONS: Unilateral occlusion of the common carotid artery in albino rats resulted in cognitive impairments, damage of neurons in the most vulnerable areas of the cortex of the cerebral hemispheres and hippocampus predominantly on the ipsilateral side. Cognitive impairments and ischaemic lesions of the brain structures are induced by endothelial dysfunction, enhanced desquamation of endotheliocytes and prevalence of vasoconstrictive reactions resulting from decreased production of the major vasorelaxing factor - nitric oxide.
  • NSAID associated bilateral renal infarctions: a case report. [Journal Article]
    Int J Nephrol Renovasc Dis 2019; 12:177-181Jeon Y, Lis JB, Chang WG
  • Renal infarctions (RIs) are caused by interruptions in the renal arterial blood flow. RIs are generally considered to be rare, however we present the case of a 37 year old woman whose renal infarction was likely due to the vasoconstrictive effects of non-steroidal anti-inflammatory drugs. Although high-dose non-steroidal anti-inflammatory drugs (NSAIDs) are known to cause a decrease in renal perf…
  • Sodium-activated potassium channels moderate excitability in vascular smooth muscle. [Journal Article]
    J Physiol 2019Li P, Halabi CM, … Salkoff L
  • CONCLUSIONS: We report that a sodium-activated potassium current, IKNa , has been inadvertently overlooked in both conduit and resistance arterial smooth muscle cells. IKNa is a major K+ resting conductance and is absent in cells of IKNa knockout (KO) mice. The phenotype of the IKNa KO is mild hypertension, although KO mice react more strongly than wild-type with raised blood pressure when challenged with vasoconstrictive agents. IKNa is negatively regulated by angiotensin II acting through Gαq protein-coupled receptors. In current clamp, KO arterial smooth muscle cells have easily evoked Ca2+ -dependent action potentials.
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