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4,290 results
  • Normal-Appearing Cerebellar Damage in Neuromyelitis Optica Spectrum Disorder. [Journal Article]
  • AAAJNR Am J Neuroradiol 2019 Jun 20
  • Sun J, Zhang N, … Yu C
  • CONCLUSIONS: We have shown that the damage in cerebellar normal-appearing white matter and normal-appearing gray matter is associated with aquaporin-4-mediated primary damage or axonal degeneration secondary to spinal lesions or both. The etiologic classifications of substructure-specific occult cerebellar damage may facilitate developing neuroimaging markers for assessing the severity and the results of therapy of neuromyelitis optica spectrum disorder occult cerebellar damage.
  • Resting-State Brain Network Dysfunctions Associated With Visuomotor Impairments in Autism Spectrum Disorder. [Journal Article]
  • FIFront Integr Neurosci 2019; 13:17
  • Wang Z, Wang Y, … Mosconi MW
  • CONCLUSIONS: Together, these findings suggest that increased resting oscillations within visuomotor networks in ASD are associated with more severe deficits in controlling variability during precision visuomotor behavior. Differences between individuals with ASD and TD controls in the topography of networks showing relationships to visuomotor behavior suggest atypical patterns of cerebellar-cortical specialization and connectivity in ASD that underlies previously documented visuomotor deficits.
  • GeneReviews®: Huppke-Brendel Syndrome [BOOK]
  • BOOKUniversity of Washington, Seattle: Seattle (WA)
  • Adam MP, Ardinger HH, … Amemiya ABindu PS, Chiplunkar S, … Taly A
  • Huppke-Brendel syndrome (HBS) is characterized by bilateral congenital cataracts, sensorineural hearing loss, and severe developmental delay. To date, six individuals with HBS have been reported in t…
  • Alteration of cerebello-thalamocortical spontaneous low-frequency oscillations in juvenile myoclonic epilepsy. [Journal Article]
  • ANActa Neurol Scand 2019 Jun 08
  • Kim JH, Kim JB, Suh SI
  • CONCLUSIONS: We have shown alterations of low-frequency oscillations in the thalamus, insular cortex/inferior frontal gyrus, and cerebellum in patients with JME, implicating cerebello-thalamocortical network abnormality in the pathophysiology underlying JME. Our results could further support the recent concept that JME is a network epilepsy involving specific cortical and subcortical structures, especially the cerebello-thalamocortical network.
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