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22 results
  • Mechanism of lymphocytic choriomeningitis virus entry into cells. [Journal Article]
    Virology 1994; 198(1):1-9Borrow P, Oldstone MB
  • The path that the arenavirus lymphocytic choriomeningitis virus (LCMV) uses to enter rodent fibroblastic cell lines was dissected by infectivity and inhibition studies and immunoelectron microscopy. Lysosomotropic weak bases (chloroquine and ammonium chloride) and carboxylic ionophores (monensin and nigericin) inhibited virus entry, assessed as virus nucleoprotein expression at early times post-i…
  • Effect of amantadine on rhabdovirus infection. [Journal Article]
    Drugs Exp Clin Res 1985; 11(1):69-74Superti F, Seganti L, … Orsi N
  • Enveloped viruses enter host cells by fusion or viropexis. The latter mechanism is the prevalent entry pathway of rhabdoviruses into susceptible cells. Amantadine, a lysosomotropic agent, inhibits the multiplication of various groups of viruses. The effect of this drug was investigated on vesicular stomatitis virus and rabies fixed virus strain replication in fibroblasts. Amantadine was added to …
  • Entry of mouse hepatitis virus 3 into cells. [Journal Article]
    J Gen Virol 1984; 65 ( Pt 1):227-31Krzystyniak K, Dupuy JM
  • The involvement of lysosomes in infection by mouse hepatitis virus 3 (MHV3) was studied. L cells were infected with MHV3 in the presence of NH4Cl or chloroquine, weak bases which increase the intralysosomal pH and impair lysosomal functions. NH4Cl significantly inhibited virus-induced cytopathic effects and MHV3 replication, but did not prevent the attachment of 3H-labelled virus. No inhibition o…
  • Two modes of entry of reovirus particles into L cells. [Journal Article]
    J Gen Virol 1979; 45(1):161-70Borsa J, Morash BD, … Szekely JG
  • Evidence is presented supporting the hypothesis that reovirus intermediate subviral particles (ISVP), which show increased infectivity relative to intact virions, can gain entry into host L cells by two alternative pathways. One pathway is by the process of viropexis, involving phagocytic vacuoles. A second entry pathway is via direct penetration of the plasma membrane of the cell, without involv…
  • Studies on the mechanism of influenza virus entry into cells. [Journal Article]
    J Gen Virol 1979; 43(1):223-9Patterson S, Oxford JS, Dourmashkin RR
  • Inhibitors of glycolysis, oxidative phosphorylation, protein synthesis, membrane Na&-K& transport and microfilament and microtubule function have been employed to elucidate the mechanism of influenza virus uptake by CAM and CEF cells. Electron microscopy demonstrated uptake of virus by viropexis in the presence of all these inhibitors. Utilizing a pulse labelling technique, virus entering CEF cel…
  • Studies of membrane fusion. IV. Fusion of HeLa cells with Sendai virus. [Journal Article]
    J Cell Sci 1979; 36:73-84Knutton S
  • The Sendai virus-induced fusion of HeLa cells has been studied by freeze-fracture electron microscopy. Freeze-fracture observations confirm previous scanning electron-microscope studies (1977) and show that at 4 degrees C virus particles bind to the cell surface and that cell agglutination results from the crosslinking by virus particles of microvilli on adjacent cells. Incubation at 37 degrees C…
  • C-type virus-lymphocyte interactions in developing mouse thymus. [Journal Article]
    Am J Anat 1978; 153(1):165-70Koppenheffer TL, Phillips JH, Vankin GL
  • The appearance of C-type virus particles in thymus cells of Swiss mouse embryos, 11.5 to 15.5 days post-conception age (PCA), was studied with the electron microscope. In thymic rudiments of all specimens examined, virus particles were seen in epithelial cytoplasm, budding from epithelial cell surfaces and in extracellular spaces. Lymphoid cells were first seen in thymic rudiments of 13.5 days PC…
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