The role of allergens in atopic dermatitis.Clin Rev Allergy. 1993; 11(4):471-90.CR
Abstract
AD is a disorder that affects up to 12% of the pediatric population. This disease is multifactorial and encompasses a wide array of etiologic factors. Strong evidence has existed in the literature over the past century for the role of inhalant and food allergies in the pathogenesis of AD. Much work is currently ongoing to delineate the role of individual cellular components, cytokines, and other mediators in the pathogenesis of AD. The answers to these questions, as well as a more comprehensive understanding of hereditary factors, will provide key information to our overall understanding of AD and our ability to treat patients with this disease more effectively in the future.
Pub Type(s)
Journal Article
Review
Language
eng
PubMed ID
8143262
Citation
Jones, S M., and H A. Sampson. "The Role of Allergens in Atopic Dermatitis." Clinical Reviews in Allergy, vol. 11, no. 4, 1993, pp. 471-90.
Jones SM, Sampson HA. The role of allergens in atopic dermatitis. Clin Rev Allergy. 1993;11(4):471-90.
Jones, S. M., & Sampson, H. A. (1993). The role of allergens in atopic dermatitis. Clinical Reviews in Allergy, 11(4), 471-90.
Jones SM, Sampson HA. The Role of Allergens in Atopic Dermatitis. Clin Rev Allergy. 1993;11(4):471-90. PubMed PMID: 8143262.
* Article titles in AMA citation format should be in sentence-case
TY - JOUR
T1 - The role of allergens in atopic dermatitis.
AU - Jones,S M,
AU - Sampson,H A,
PY - 1993/1/1/pubmed
PY - 1993/1/1/medline
PY - 1993/1/1/entrez
SP - 471
EP - 90
JF - Clinical reviews in allergy
JO - Clin Rev Allergy
VL - 11
IS - 4
N2 - AD is a disorder that affects up to 12% of the pediatric population. This disease is multifactorial and encompasses a wide array of etiologic factors. Strong evidence has existed in the literature over the past century for the role of inhalant and food allergies in the pathogenesis of AD. Much work is currently ongoing to delineate the role of individual cellular components, cytokines, and other mediators in the pathogenesis of AD. The answers to these questions, as well as a more comprehensive understanding of hereditary factors, will provide key information to our overall understanding of AD and our ability to treat patients with this disease more effectively in the future.
SN - 0731-8235
UR - https://www.unboundmedicine.com/medline/citation/8143262/full_citation
L2 - http://www.diseaseinfosearch.org/result/9652
DB - PRIME
DP - Unbound Medicine
ER -
